Acute anaphylaxis

Theory

Immunoglobulin E mediated response to antigenic stimulus involving MAST cell degranulation.

Histamine release causes generalised systemic response.

Airway – angio-oedema and laryngeal oedema leading to progressive and rapid airway obstruction and respiratory arrest.

Breathing – bronchospasm resulting in progressive obstruction and increased work of breathing.

Circulation – vasodilatation with resultant hypotension, tachycardia and cardiovascular collapse – (shock).

The combination, the rate of progression, and the severity of symptoms are variable; therefore, all patients with anaphylaxis who are still deteriorating are potentially at risk of death and require prompt treatment.

Immediate intern management

Attend patient and make rapid assessment.

Patient unconscious

  1. Tell nursing staff to call Code Blue then move to head end of bed.
  2. Perform triple manoeuvre – chin lift, jaw thrust, head tilt.
  3. Bag and mask patient with oxygen. (Be careful with instrumenting an airway in anaphylaxis – can make it worse. Secure airway with bag and mask.)
  4. When help arrives obtain IV access, send off blood tests.
  5. Give IV adrenaline one ampoule in 500 ml Gelofusine stat.

Patient conscious

  1. Tell nursing staff to call code MET.

Then:

  1. Give oxygen by mask.
  2. Get crash trolley, obtain IV access and send off blood tests (FBE, U&E, LFT, CRP).

Patient normotensive

  1. Give 300 micrograms adrenaline S/C or IM.
  2. Give 500 ml Gelofusine stat.
    Help should arrive by now.

Patient hypotensive

Give IV adrenaline 50 micrograms bolus and repeat every two to three minutes.

If the patient has known anaphylaxis, an Epi-Pen (subcutaneous adrenaline pen) may be available for use in an emergency.

Clinical features (obtain rapidly)

History

Examination

Other (obtain later)

Risk factors: past history anaphylaxis, family history, asthmatic

Suspect the diagnosis when there are two or more of:

Further definitive management

Airway

Breathing

Circulation

Other

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